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Corrigendum for Paria et al., Am J Physiol Lung Cell Mol Physiol 287 (6) L1303-L1313.
Am J Physiol Lung Cell Mol Physiol 297: L204, 2009; doi:10.1152/ajplung.zh5-5479-corr.2009
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Corrigendum

Volume 287, December 2004 Paria BC et al. Tumor necrosis factor-{alpha}-induced TRPC1 expression amplifies store-operated Ca2+ influx and endothelial permeability. Am J Physiol Lung Cell Mol Physiol 287: L1303–L1313, 2004; doi:10.1152/ajplung.00240.2004; http://ajplung.physiology.org/cgi/content/full/287/6/L1303.

We now substitute the original incorrect electrophysiological recording shown in Fig. 5B with the correct tracing from our data files. The results and conclusions of this study stand. The authors apologize for the error.


Figure 5
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Fig. 5. Effect of increased TRPC1 expression on current through store-operated channels. B: human dermal microvascular endothelial cells were transfected with TRPC1 cDNA. Isolated single cells were voltage clamped to a potential of –50 mV using a patch pipette containing inositol 1,4,5-trisphosphate plus dibenzohydroquinone to deplete intracellular calcium stores (endoplasmic reticulum). Current through SOCs rapidly developed as shown. La3+ (100 µM) was added where indicated to block the current.

 





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